Qing-Xin-Jie-Yu granule prevents myocardial infarction-induced apoptosis via inhibition of p38 MAPK pathway

Key Points

Cardiac function improves significantly post-treatment with QXJYG, indicating effective cardioprotection.
Apoptosis regulation via the p38 MAPK pathway marks a notable mechanism in this cardioprotective effect.
The study includes both in vivo and in vitro assessments for comprehensive evaluation.
Implications highlight potential therapeutic uses for QXJYG in managing myocardial infarction.

Abstract

This study demonstrates that QXJYG exerts cardioprotective effects against MI by improving cardiac function, reducing oxidative stress, and inhibiting apoptosis through the regulation of the p38 MAPK signaling pathway both in vivo and in vitro.

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Qing-Xin-Jie-Yu granule prevents myocardial infarction-induced apoptosis via inhibition of p38 MAPK pathway

Key Points

Abstract

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