Virulent and attenuated DHAV-3 elicit divergent host transcriptomic responses in duck liver, highlighting IFN-I signaling as a central axis in antiviral immunity.
IFN-I signaling serves as a central axis in the antiviral immune response to DHAV-3 in ducks.
A Correction on: Fu Q, Jiao W, Wan C, Wang W, Su J, Liu X, Cheng L, Chen H, Liu J, Huang Y and Fu G (2026) Comparative analysis of ducks liver gene expressions infected with virulent or attenuated DHAV-3 reveals divergent host responses to viruses of different virulence. Front. Microbiol. 17:1740464. doi: 10.3389/fmicb.2026.1740464A correction has been made to the section Abstract: Discussion: "These findings delineate divergent host transcriptomic responses to virulent vs. attenuated DHAV-3 and highlight IFN-I signaling as a central axis in antiviral immunity."The original version of this article has been updated.
Frontiers Production Office (Tue,) conducted a other in DHAV-3 infection. Virulent DHAV-3 vs. Attenuated DHAV-3 was evaluated on Host transcriptomic responses. Virulent and attenuated DHAV-3 elicit divergent host transcriptomic responses in duck liver, highlighting IFN-I signaling as a central axis in antiviral immunity.