COL23A1 promotes tumor progression and glycolytic reprogramming in endometrial cancer via METTL14-m6A-YTHDF1 axis

Key Points

• Tumor progression is significantly associated with COL23A1 levels in endometrial cancer.
• High COL23A1 expression correlates with increased glycolytic reprogramming, supporting cancer metabolism.
• Analysis of molecular interactions highlights the METTL14-m6A-YTHDF1 axis as a key regulatory pathway.
• Findings may lead to targeted therapies focusing on COL23A1 and related pathways for endometrial cancer.