ABSTRACT While growth hormone therapy has been associated with the potential risk of upper airway obstruction and hypertrophy, it may also cause glottic or subglottic edema and stridor, even in patients without adenotonsillar tissue. Clinicians should consider medication‐induced airway changes in Prader–Willi Syndrome patients with complex airway histories to optimize management.
Saad et al. (Sun,) studied this question.