This manuscript presents a hypothesis exploring the role of environmental spectral radiation in biological systems, with particular focus on its potential influence on genomic stability, ageing, and evolutionary processes. While existing models of ageing emphasise intrinsic mechanisms such as DNA damage accumulation, oxidative stress, and telomere attrition, this work proposes that continuous interaction with environmental radiation may represent an additional, underexplored determinant of biological function. The hypothesis considers whether specific components of the radiation spectrum, whic are beyond currently recognised pathways such as vitamin D synthesis and photosynthesis, whether they may contribute to maintenance of cellular integrity through as yet unidentified biological mechanisms. It further explores the possibility that variation in environmental radiation exposure could influence evolutionary trajectories by modulating mutation patterns and repair processes. Analogies from natural systems, including the phenomenon of chlorosis in light-deprived environments, are used to frame the broader concept that deprivation of certain spectral inputs may contribute to biological decline. The manuscript integrates perspectives from evolutionary biology, developmental biology, and biophysics to propose a testable framework for future experimental investigation. This work is intended as a theoretical contribution aimed at stimulating discussion and guiding empirical research.
Rajinder Pal Singh (Tue,) studied this question.