ions that cause membrane disruption and internal tissue damage. The enhanced toxicity of ZnO-C1 is attributed to its smaller size, larger surface area, and improved dispersion stability, which facilitate greater ROS generation and ionic dissolution. These findings demonstrate a distinct UV-dependent mode of action and highlight the potential of nanoscale ZnO as a photoactivated larvicidal agent against Ae. aegypti.
Bakhori et al. (Fri,) studied this question.