Why the study?
How changes in visceral adipose tissue modulate circulating inflammatory proteins and interact with glycemic control remains unclear.
Are longitudinal changes in visceral adipose tissue and glycemic control associated with changes in inflammatory proteomic profiles in middle- to older-aged adults?
Population
562 middle- to older-aged US adults in the VITAL study
Comparison
Longitudinal changes in VAT mass and glycemic traits
Design
Prospective cohort study
Follow-up
2 years
Key result
Longitudinal gain in visceral adipose tissue over 2 years was significantly associated with concurrent changes in 34 inflammatory proteins (FDR<0.05), linking VAT change to metabolic dysregulation.
Authors
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Reassures clinicians on short-term VAT and glycemic trajectories in at-risk adults; leaves open mediating role of inflammatory proteomics.
Cohort (n=562)
Are longitudinal changes in visceral adipose tissue and glycemic control associated with changes in inflammatory proteomic profiles in middle- to older-aged adults?
p-value: p=<0.05 (FDR)
Longitudinal changes in visceral adiposity and glycemic control are associated with distinct inflammatory proteomic changes, highlighting a biological link between VAT, metabolic dysregulation, and inflammation.
Wang et al. (2026) conducted a cohort in Obesity and risk of T2D and CVD (n=562). Longitudinal changes in visceral adipose tissue (VAT) was evaluated on Associations between 2-year changes in VAT and individual inflammatory proteins (p=<0.05 (FDR)). Longitudinal gain in visceral adipose tissue over 2 years was significantly associated with concurrent changes in 34 inflammatory proteins (FDR<0.05), linking VAT change to metabolic dysregulation.