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March 3, 2026Experimental & Molecular MedicineOpen Access

Impaired insulin secretion via the Wnt5a/β-catenin pathway contributes to diabetes development in pancreatic cancer

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Authors

MLMinyoung LeeHPHo Seon ParkHKHyung Sun Kim

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Overview

Observational analysis finds impaired insulin secretion in pancreatic cancer patients, indicating a potential biomarker for diabetes development.

Key Points

  • Patients with pancreatic cancer demonstrated severe hyperglycemia with impaired insulin secretion before surgery, suggesting a critical link to diabetes.
  • In a cohort of 160 patients, those with pancreatic ductal adenocarcinoma showed greater insulin deficiency compared to counterparts without cancer.
  • Assessment using oral glucose tolerance tests revealed significant differences in glucometabolic parameters pre- and post-surgery among patients.
  • These findings support the hypothesis that alterations in the Wnt5a/beta-catenin pathway may drive diabetes risk in pancreatic cancer patients.

Cite This Study

Lee et al. (2026) studied this question.

synapsesocial.com/papers/69a75afbc6e9836116a21849https://doi.org/10.1038/s12276-025-01625-8
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