Why the study?
Evaluating the relationship between CMR-derived myocardial strain and peak oxygen uptake from CPET could help assess ventricular dysfunction and its impact on functional capacity in patients with HCM.
Does CMR-derived myocardial strain correlate with functional impairment (VO₂ peak) in patients with hypertrophic cardiomyopathy?
Population
32 HCM patients diagnosed between January 2017 and April 2023
Design
Cohort study
Key result
CMR-derived myocardial strain correlates with LV mass index (β=0.08, p=0.001) and fibrosis extent (β=9.8, p=0.022) but not with VO2 peak in HCM patients.
Authors
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Strain fails to track exercise capacity in HCM despite structural links; leaves open its incremental prognostic role.
Does CMR-derived myocardial strain correlate with functional impairment (VO₂ peak) in patients with hypertrophic cardiomyopathy?
In patients with hypertrophic cardiomyopathy, CMR-derived myocardial strain correlates with LV mass and fibrosis but does not correlate with functional exercise capacity (VO₂ peak).
Cantora et al. (2025) studied this question. CMR-derived myocardial strain correlates with LV mass index (β=0.08, p=0.001) and fibrosis extent (β=9.8, p=0.022) but not with VO2 peak in HCM patients.