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February 19, 2026Retina1 citations

Role of Inflammation in Atrophy Development and Progression in Pathologic Myopia

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NGNoa GileadYCYu Jeat ChongJLJoshua Lim

Key Points

  • This research aims to explore how prior retinal inflammation affects atrophy progression in pathologic myopia.
  • Retrospective case series of 21 inflamed eyes and 26 noninflamed controls
  • Assessment of annual atrophy-area change using multimodal imaging
  • Group comparisons through Welch t tests and Fisher exact tests
  • Age-adjusted linear regression to identify associations
  • Inflamed eyes displayed a faster atrophy progression rate of 0.76 mm²/year compared to 0.38 mm²/year in controls (P = 0.03)
  • 100% of inflamed eyes developed new atrophy versus 36.4% in controls (P < 0.01)
  • Atrophy activity was higher in inflamed eyes (100%) compared to controls (73.1%) (P < 0.01)
  • Multifocal atrophy occurred more frequently in inflamed eyes (71.4%) than in controls (23.1%) (P < 0.01)
  • Inflammation was independently linked to faster atrophy expansion (β = 0.41 mm²/year; P = 0.033)

Abstract

Purpose: To describe the progression of atrophy in pathologic myopia eyes with prior retinal inflammatory lesions consistent with punctate inner choroidopathy. Methods: Retrospective case series of 21 eyes with pathologic myopia and resolved retinal inflammatory lesions compared with 26 noninflamed controls (14 fellow eyes; 12 external myopic eyes with pre-existing patchy atrophy). The primary outcome was annual atrophy-area change (mm 2 /year) on multimodal imaging. Secondary outcomes included incident/enlarging atrophy, multifocal versus unifocal appearance, and META-PM category progression. Group comparisons used Welch t tests and Fisher exact tests; an age-adjusted linear regression examined independent associations. Results: Over 5.2 ± 3.2 years in inflamed eyes and 5.3 ± 2.4 years in controls, patchy atrophy progressed faster with inflammation (0.76 ± 0.67 vs. 0.38 ± 0.47 mm 2 /year; P = 0.03). Among eyes starting in myopic macular degeneration Category 1 to 2, incident atrophy occurred in 14 of 14 (100%) inflamed versus 4 of 11 (36.4%) controls ( P < 0.01). Overall atrophy activity (new or enlarging) was 21 of 21 (100%) in inflamed eyes versus 19 of 26 (73.1%) controls ( P < 0.01). Multifocal atrophy was more frequent with inflammation (15/21, 71.4%) than in controls (6/26, 23.1%; P < 0.01). In age-adjusted regression, inflammation remained independently associated with faster atrophy expansion (β = 0.41 mm 2 /year; 95% CI, 0.04–0.78; P = 0.033). Conclusion: In pathologic myopia, prior retinal inflammation is independently associated with more frequent multifocal involvement, higher risk of incident atrophy, and a substantially faster atrophy-expansion. Beyond mechanical factors, inflammation seems to be a key driver of atrophic progression in this population.

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Cite This Study

Gilead et al. (2026) studied this question.

synapsesocial.com/papers/6996a887ecb39a600b3ef54ahttps://doi.org/10.1097/iae.0000000000004697
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