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February 24, 2026Alzheimer s & Dementia0 citationsOpen Access

Association of apolipoprotein E variants on Alzheimer's disease in Latin America: A systematic review and meta‐analysis

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POPaulina OrellanaACAriel CaviedesLGLiset Gonzalez

Key Points

  • To investigate the association of apolipoprotein E (APOE) variants with Alzheimer's disease (AD) risk in Latin America.
  • Conducted a systematic review and meta-analysis of 35 studies from 11 Latin American and Caribbean countries.
  • Included 3206 patients diagnosed with Alzheimer's disease and 5515 controls.
  • Analyzed the relationship between different APOE alleles and AD risk using odds ratios.
  • The APOE ε4 allele is associated with a significant increased risk of Alzheimer's disease (OR = 3.25).
  • Homozygous ε4/ε4 carriers have a notably higher risk of AD (OR = 6.84).
  • Heterozygous ε3/ε4 carriers present a moderate risk (OR = 2.59).
  • Variability across countries, with Ecuador reporting the highest risk associated with ε4/ε4 (OR = 13.29).

Abstract

Abstract The apolipoprotein E ( APOE ) ε4 allele represents the strongest genetic risk factor for Alzheimer's disease (AD), but its role in genetically diverse Latin American and Caribbean (LAC) populations is underexplored. We conducted a meta‐analysis of 35 studies from 11 LAC countries, encompassing 3206 patients with AD and 5515 controls. The ε4 allele demonstrated significant association with increased AD risk (odds ratio OR = 3.25, 95% confidence interval 2.82–3.76), while ε3 showed lower odds (0.42, 0.37–0.48). Homozygous ε4/ε4 carriers had elevated risk (6.84, 5.09–9.19), and heterozygous ε3/ε4 carriers showed moderate risk (2.59, 2.31–2.91). Country‐level analyses revealed variability, with Ecuador showing the highest OR for ε4/ε4 (13.29, 1.56–113.4). These results confirm APOE ε4 as a major AD risk factor in LAC populations and highlight regional differences relevant to precision medicine.

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Cite This Study

Orellana et al. (2026) studied this question.

synapsesocial.com/papers/699d3fe6de8e28729cf64c07https://doi.org/10.1002/alz.71224
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