血管平滑筋細胞RNA結合タンパク質U2AF2は、C1qbp発現を調節することで銅による死を誘導し、動脈硬化の進展を遅延させる

Yu et al. (Wed,) はこの問題を研究しました。

www.synapsesocial.com/papers/69a75bb5c6e9836116a23869 — DOI: https://doi.org/10.1186/s40659-026-00672-3

Also consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1. Complement C1q Binding Protein (C1QBP): Physiological Functions, Mutation-Associated Mitochondrial Cardiomyopathy and Current Disease Models· 2022 · 26 citations
2. Gas Chromatography–Mass Spectrometry Reveals Stage-Specific Metabolic Signatures of Ankylosing Spondylitis· 2023 · 6 citations
3. Evaluation the role of cuproptosis-related genes in the pathogenesis, diagnosis and molecular subtypes identification of atherosclerosis· 2023 · 12 citations
4. The Mitochondrial Protein C1QBP Promotes Hepatocellular Carcinoma Progression by Enhancing Cell Survival, Migration and Invasion· 2022 · 16 citations
5. NEAT1 regulates VSMC differentiation and calcification in as long noncoding RNA NEAT1 enhances phenotypic and osteogenic switching of vascular smooth muscle cells in atherosclerosis via scaffolding EZH2· 2024 · 15 citations