Correction: Reciprocal regulation of oxidative stress and mitochondrial fission augments parvalbumin downregulation through CDK5-DRP1- and GPx1-NF-κB signaling pathways

Key Points

• Parvalbumin downregulation is enhanced through oxidative stress and mitochondrial fission pathways.
• Key metrics indicate a significant involvement of cdk5 and drp1 in this process, leading to altered cell function.
• Observational analysis examines the relationship between oxidative stress and signaling pathways affecting parvalbumin.
• Understanding this reciprocal regulation may inform strategies for cellular health interventions.