These findings demonstrate that PFAS-induced pro-inflammatory cytokines is mediated, at least in part, through PPARγ signaling, and that pharmacological activation of PPARγ signaling can attenuate PFAS-triggered pro-inflammatory cytokine responses in lung epithelial cells.
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Shaikh et al. (Tue,) studied this question.
www.synapsesocial.com/papers/69a76068c6e9836116a2d1ff — DOI: https://doi.org/10.3389/fphar.2026.1779345
Sadiya Bi Shaikh
Md Imam Faizan
Khursheed Ul Islam
Frontiers in Pharmacology
University of Rochester Medical Center
Harbor–UCLA Medical Center
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