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April 1, 2026Frontiers in Bioscience-Landmark1 citationsOpen Access

The Dual Role of DNA Hypermethylation and Hypomethylation in Colorectal and Gastric Tumorigenesis: Mechanisms and Non-Invasive Biomarker

LBLong BaiXYXiaoli YanZWZi Wang

Key Points

  • The research explores how DNA hypermethylation and hypomethylation contribute to tumorigenesis in colorectal and gastric cancers.
  • Reviewed molecular mechanisms of DNA methylation
  • Analyzed the effect of methylation on tumor suppressor genes and oncogenes
  • Evaluated clinical significance as a non-invasive biomarker
  • Hypermethylation silences tumor suppressor genes, promoting tumor initiation
  • Global hypomethylation leads to genomic instability and oncogene activation
  • Potential for DNA methylation patterns to serve as non-invasive biomarkers for diagnosis

Abstract

DNA methylation is a key epigenetic modification catalyzed by DNA methyltransferases (DNMTs) and predominantly occurs at cytosine-phosphate-guanine (CpG) islands, which are often located in gene promoter regions. Hypermethylation of CpG islands within gene promoters can silence tumor suppressor gene expression, thereby disrupting normal cellular functions, including maintenance of genomic stability and regulation of cell growth, and contributing to tumor initiation and progression. In contrast, global hypomethylation may promote genomic instability and oncogene activation. This review discusses the molecular mechanisms underlying DNA methylation and evaluates its functional and clinical significance in colorectal and gastric cancers, with emphasis on its potential application as a noninvasive biomarker for diagnosis.

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Cite This Study

Bai et al. (2026) studied this question.

synapsesocial.com/papers/69cd7b695652765b073a9701https://doi.org/10.31083/fbl46159
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