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April 8, 2026Macromol—A Journal of Macromolecular Research0 citationsOpen Access

Anti-Inflammatory Activities of Zophobas morio Larvae Lipids on Lipopolysaccharide-Induced Activated Macrophages: Reprogramming Macrophage Polarization and Attenuating Oxidative Stress

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JPJu-Hwi ParkHCHa-Seong ChoJNJu‐Ock Nam

Key Points

  • This research aims to evaluate the anti-inflammatory effects of lipids from Zophobas morio on macrophages activated by lipopolysaccharides.
  • Analyzed the fatty acid composition of Zophobas morio lipids (ZMLs) using FT-IR and 1H-NMR techniques.
  • Conducted thermogravimetric analysis to assess thermal stability.
  • Examined the effects of ZML on macrophage polarization and inflammatory responses in laboratory experiments.
  • ZML shows a higher proportion of unsaturated fatty acids compared to saturated ones.
  • ZML significantly inhibits lipopolysaccharide-induced M1 macrophage polarization by suppressing NF-κB and MAPK signaling pathways.
  • ZML activates the Nrf2-mediated antioxidant pathway, alleviating oxidative stress.

Abstract

This study investigated the structural and anti-inflammatory properties of Zophobas morio lipids (ZMLs). The fatty acid (FAs) composition showed a higher proportion of unsaturated FAs, mainly consisting of oleic (30.30%) and linoleic acids (20.05%), than saturated FAs, including palmitic (24.80%) and stearic acids (12.96%). In addition, FT-IR and 1H-NMR analyses confirmed that ZML possessed a typical triglyceride structure, with long-chain alkyl groups. Thermogravimetric analysis (TGA) indicated that ZML exhibited high thermal stability, with a degradation peak at 369 °C. Differential scanning calorimetry (DSC) displayed a thermal transition at −8 °C, corresponding to the crystallization of unsaturated FAs in ZML. ZML significantly inhibits lipopolysaccharide (LPS)-induced pro-inflammatory M1 macrophage polarization by suppressing nuclear factor κB (NF-κB) and mitogen-activated protein kinase (MAPK) signaling pathways, thereby attenuating the expression of inflammatory mediators. Additionally, ZML alleviated inflammatory oxidative stress by activating the nuclear factor erythroid 2-related factor 2 (Nrf2)-mediated antioxidant pathway. Notably, ZML not only induced M2 macrophage polarization in quiescent macrophages but also reprogrammed M1 macrophages toward the anti-inflammatory M2 phenotype. These findings suggest that ZML is a natural nutritional lipid source and a potential therapeutic agent for modulating inflammatory response.

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Cite This Study

Park et al. (2026) studied this question.

synapsesocial.com/papers/69d5f14b74eaea4b11a7adbehttps://doi.org/10.3390/macromol6020021
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