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April 24, 2026JCO Precision Oncology0 citations

Targeted Molecular Profiling of Circulating Cell-Free DNA in Patients With Gastroesophageal Adenocarcinoma

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SCSamuel L. CytrynRBRaktim BorpatragohainKCKarmelina Charalambous

Key Points

  • The study aims to characterize the genomic landscape of gastroesophageal adenocarcinoma through analysis of circulating cell-free DNA and tissue samples.
  • Conducted a biospecimen-collection study between 2017-2022 analyzing cfDNA and tissue NGS.
  • Utilized a clinically validated 129-gene NGS assay (MSK-ACCESS) for cfDNA analysis.
  • Performed matched-tumor NGS on 154 patients to assess predictive value of ctDNA.
  • Similar frequencies of common alterations were found between cfDNA and tissue NGS, but lower detection of copy-number alterations in plasma.
  • ctDNA detection was associated with improved overall survival and recurrence-free survival in localized disease.
  • Persistence of ctDNA indicated worse survival outcomes and predicted recurrence earlier than imaging.

Abstract

PURPOSE We sought to characterize the genomic landscape of gastroesophageal adenocarcinoma (GEA) using next-generation sequencing (NGS) of circulating cell-free DNA (cfDNA), compare the genomic profile to tissue NGS, and determine the association of circulating tumor DNA (ctDNA) detection with clinical outcomes. METHODS In this prospective biospecimen-collection study (2017-2022), cfDNA was analyzed using a clinically validated 129-gene tumor-informed NGS assay Memorial Sloan Kettering-Analysis of Circulating cfDNA to Evaluate Somatic Status (MSK-ACCESS); tissue-NGS was performed using Memorial Sloan Kettering-Integrated Mutation Profiling of Actionable Cancer Targets. RESULTS In total, 169 patients with GEA underwent baseline molecular profiling of cfDNA; 93 had localized disease, 55 had untreated metastatic disease, and 21 had progressive metastatic disease. The frequency of the most common alterations was similar between platforms, although copy-number alterations were detected less frequently in plasma than in tissue (10% v 40%; P < .001). Matched-tumor NGS was conducted in 154 patients (91%). Baseline ctDNA detection and on-treatment clearance were associated with improved overall survival and recurrence-free survival in patients with localized disease and progression-free survival in patients with metastatic disease. Baseline ctDNA detection was a stronger predictor of overall survival than American Joint Committee on Cancer (AJCC) clinical stage and positively predicted lymph node metastasis in 87% of cases. Trends in variant allele fractions correlated with pathologic responses to neoadjuvant therapy more closely than fluorodeoxyglucose-positron emission tomography standard uptake values. Persistence or resurgence of ctDNA was associated with worse survival and forecasted recurrent disease a median of 7 months sooner than imaging. CONCLUSION cfDNA analysis using MSK-ACCESS in patients with GEA is a valuable adjunctive clinical tool that enhances molecular profiling, prognostication, treatment response assessment, and detection of recurrent disease in conjunction with tissue NGS, imaging, and AJCC clinical staging criteria.

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Cite This Study

Cytryn et al. (2026) studied this question.

synapsesocial.com/papers/69eb0a66553a5433e34b47fahttps://doi.org/10.1200/po-25-01069
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