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May 6, 2026World Journal of Clinical Medicine0 citations

Hapten Immunetherapy for Liver Cancer HBVsup+/sup 40 Years and Restores Normal HBVsup-/sup

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BYBaofa YuJZJ Y ZhangYHYan Han

Key Points

  • To evaluate the impact of hapten immunotherapy on enhancing the immune response in liver cancer patients with chronic HBV infection.
  • Utilized single-cell RNA sequencing to assess tumor changes post-treatment
  • Evaluated immune cell profiles and gene expression related to MHC pathways after therapy
  • Analyzed the reduction of HBV-infection pathways and immune cell populations.
  • Significant increase in MHC II signaling pathway-related genes in B cells post-treatment
  • Reduction in HBV-infection scores and associated immune cells
  • Patients showed a drop in HBV DNA titer to zero and improved control of HCC masses.

Abstract

Chronic hepatitis B virus (HBV) infection has considered a major risk factor for the development and progression of hepatocellular carcinoma (HCC), long-term lack of effective treatment worldwide. Hapten enhanced intratumoral chemotherap may provide a revival effect of immune system to against cancer cell of HCC and HBV. Methods. Single-cell RNA sequencing (scRNA-seq) was used to analyze the changes at the molecular level of tumor reflection from the untreated tumor after HEIC treated a few multi-focal HCC tumors. Results. After treatment with major tumors, both BMEM and Naive B cells, significantly increased MHC II signaling pathway-related genes, while plasma cells upregulated MHC I-related genes. Fibroblast, γδT/NK cells and Neutrophils, while NKT cells, Plasma cells, Endothelial cells, and B cells and M2 Macrophage showed values of AUC less than 0.6 which indicated minor affection or possible minor contribution to the therapeutic effect. It was observed significantly reduced scores of HBV-infection pathways, the percentages of CSTA+DCs and Kupffer cells were reduced, showing accordance with the attenuated HBV caused inflammation. Patient of HCC HBV DNA titter drop to zero and got control of the multifocal masses of HCC who still live. It is first discovered hapten is modifiing neo HBsAg (HBsAg) while it is modifying the TAAs’s epitope, which return to a neo HBsAg, neu TAAs and rejuvenate a revival effects of immune system against cancer and HBV, i.e., by activating efficacy of CD4 T-cell activation and CD8 T-cell responses, thus, prolonging the survival time of patients, and maintaining the quality of life. Conclusions. This study provides new biological knowledge of therapy, hapten based intratumoral chemoimmunotherapy is playing an important role to improve neu TAAs and neo HBsAg for a revival of immune effect to control cancer cell and HBV in HCC with HBV positive.

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Cite This Study

Yu et al. (2026) studied this question.

synapsesocial.com/papers/69faa25e04f884e66b533078https://doi.org/10.57237/j.wjcm.2026.01.002
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