PulseExploreJournal ClubDebatesTrendingResearchersJournals
Instagram
HomeExploreJournal ClubTrending
Synapse
⌘+K
Synapse
May 6, 2026Obstetrics and Gynecology0 citations

Oral Contraceptive Failure With GLP-1 and Dual GLP-1/GIP Therapies: A Case Report and Systematic Review of Incretin–COC Interactions ID 3414

View Full Paper
CMCasey MoffittFCFrances E. CaseyJGJustin Gimoto

Key Points

  • To evaluate the interplay between incretin therapies and oral contraceptive failure due to pharmacokinetic alterations.
  • Conducted a systematic review of studies on incretin–COC interactions and pharmacokinetics.
  • Searched PubMed, EMBASE, Web of Science, and ClinicalTrials.gov for relevant trials and reports.
  • Included FDA reviews and package inserts related to the therapies.
  • 10 studies were reviewed, with 305 participants primarily being premenopausal women.
  • Minimal change in ethinyl estradiol exposure; however, progestin exposure was affected differently by various agents.
  • Significant reductions in LNG Cmax were observed with certain therapies, indicating altered bioavailability.

Abstract

INTRODUCTION: Glucagon-like peptide-1 receptor agonists (GLP-1 RAs) and glucose-dependent insulinotropic polypeptide (GIP)/GLP-1 agonists delay gastric emptying and may alter combined oral contraceptive (COC) pharmacokinetics (PK). We report the first documented COC failure on semaglutide and systematically review agent-specific evidence to guide counseling. METHODS: Following PRISMA guidelines, PubMed, EMBASE, Web of Science, and ClinicalTrials.gov were searched for trials, PK studies, and observational reports evaluating incretin–COC interactions (area under the curve AUC, maximum concentration Cmax, time to maximum concentration Tmax) and contraceptive outcomes. U.S. Food and Drug Administration (FDA) reviews and package inserts were included. We also report the unintended pregnancy of a 39-year-old G4P2012 with obesity, conceived after 4 months of weekly semaglutide despite monophasic ethinyl estradiol/levonorgestrel COC use. RESULTS: Of 372 records, 10 studies (305 participants; 7 randomized crossover trials, 3 FDA reviews) met criteria. Most enrolled healthy, premenopausal participants, 4 evaluated postmenopausal women. Ethinyl estradiol exposure was unchanged across studies. Progestin effects varied: semaglutide/dulaglutide showed minimal levonorgestrel (LNG) AUC change (−8% to +5%), while tirzepatide, lixisenatide, and immediate pre-COC exenatide reduced LNG Cmax by 45–66% and AUC by 18–23%. Pooled analysis demonstrated consistent Tmax delays (mean difference 1.16 hours, 95% CI, 0.10–2.22). Short-acting agents consistently produced the largest PK changes when administered before COC use. Theoretic model of delayed gastric emptying also predicts greater persistence of Tmax delays in tirzepatide despite tachyphylaxis. CONCLUSIONS/IMPLICATIONS: GLP-1 and dual GLP-1/GIP agonists generally preserve estrogen exposure but can delay and reduce progestin bioavailability. While these changes are unlikely to compromise COC efficacy in typical use, counseling should address timing strategies, backup contraception during high-risk periods, and caution with progestin-only and emergency methods.

Ask AI
Helpful
Bookmark
Share
View Full Paper

Cite This Study

Moffitt et al. (2026) studied this question.

synapsesocial.com/papers/69faa28f04f884e66b533340https://doi.org/10.1097/aog.0000000000006265.09
Ask AI
Helpful
Bookmark
Share
View Full Paper