Structural Memory Traps: Pathological Crystallization in the REK Functional and a Field-Theoretic Model of Traumatic Re-experiencing

Post-traumatic stress disorder (PTSD) is defined by a specific anomaly in memory dynamics: traumatic events are not stored as retrievable records that fade over time but as configurations that are reactivated — re-experienced — with full perceptual and affective intensity, triggered by stimuli bearing only partial structural resemblance to the original event. We address this gap within the Schrödinger–REK–RGB field-theoretic framework, introducing the concept of pathological crystallization: the regime in which the non-local memory functional εREKΨH exceeds a critical threshold λc, generating a stable attractor from which the holistic field cannot exit spontaneously. We derive λc analytically and show that it depends on three neurobiologically interpretable parameters: the coherence length ℓᵦ (hippocampal contextual embedding capacity), the integrated kernel weight K̂ (total crystallization capacity), and the crystallized density ρc (event intensity). The model provides a formal stability criterion (λc), a formal resolution condition (Eₘin), and a formal account of two distinct therapeutic mechanisms: pharmacological reduction of λREK (MDMA-assisted therapy) and graduated exposure increase of εₖ (Prolonged Exposure, EMDR). The model generates four testable predictions including a non-Markovian signature distinguishing it from existing Markovian models of intrusive memory dynamics.