A correction was issued to replace an incorrectly duplicated Tubulin loading control band in Figure 2I, which did not affect the original conclusions of the study.
db/m and db/db mice (WT and Bdh1-/-) (n=6 per group); H9C2 cells; primary cardiomyocytes isolated from fetal WT or Bdh1-/- mice
BDH1 deficiency (Bdh1-/- or siBdh1) and/or palmitic acid (PA) treatment
Wild-type (WT) mice, control siRNA (siNC), and/or vehicle (no PA) treatment
Cardiac dysfunction (LVEF, FS, E/A ratio), fibrosis, apoptosis, and inflammatory factor expressionsurrogate
This is a correction for a duplicated loading control band in a preclinical study showing that BDH1 deficiency aggravates diabetic cardiomyopathy.
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Bu-tuo Xu
Shengrong Wan
Qi Wu
Cardiovascular Diabetology
Wenzhou Medical University
Macau University of Science and Technology
Southwest Medical University
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Xu et al. (Fri,) conducted a other in diabetic cardiomyopathy. BDH1 overexpression vs. Wild type / control was evaluated. A correction was issued to replace an incorrectly duplicated Tubulin loading control band in Figure 2I, which did not affect the original conclusions of the study.
www.synapsesocial.com/papers/6a025efcc9581ed855361b2d — DOI: https://doi.org/10.1186/s12933-026-03214-z