Oral antibiotics promote triple-negative breast cancer progression by inducing gut microbiota dysbiosis and taurocholic acid accumulation

T cell infiltration and cytotoxic function in the tumor microenvironment. In summary, this study, using a mouse model, establishes a link between oral antibiotic-induced dysbiosis and accelerated breast cancer progression and proposes that TCA and its associated pathways may serve as potential targets for future TNBC therapies.