Early-life exposure to flusilazole causes transgenerational cardiac developmental defects in zebrafish and its potential causes

Key Points

• Cardiac developmental defects are observed in zebrafish due to early-life flusilazole exposure, highlighting significant risks.
• Marked structural changes were recorded in 60% of zebrafish offspring when exposed to flusilazole in early life stages.
• Analysis of embryonic development across multiple generations shows persistent defects linked to environmental exposure to flusilazole.
• Understanding the mechanism behind transgenerational effects is crucial to address potential environmental hazards.