Assessment of tissue homogenate levels of TGM1, PPL and KRT8 in a group of patients with HNSCC tumors and matched surgical margin samples

Key Points

• Impaired tgm1, ppl, and krt8 levels indicate their possible role in hnscc progression, emphasizing the need for further research.
• Key evidence suggests that altered signaling pathways of tgm1, ppl, and krt8 may affect disease outcomes in hnscc, highlighting their importance.
• Assessment of tissue homogenate levels in hnscc tumors and matched surgical margin samples reveals significant findings.
• This analysis underscores the potential clinical utility of tgm1, ppl, and krt8 as biomarkers in diagnosing hnscc.

Abstract